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Furthermore, IL-1, TNF-, and complement component subunit 1q (C1q) encourage the transformation of astrocytes into reactive astrocytes (A1), subsequently impairing the astrocytic capacity to regulate microglial activity [132]

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For example, systemic administration of 5-HT receptor agonists or fluoxetine, a selective serotonin reuptake inhibitor, increased cell proliferation and newly born neurons in the SVZ and dentate gyrus [130, 131]

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